GINGIVITIS ULCERO NECROSANTE PDF

Necrotizing ulcerative gingivitis (NUG) is a typical form of periodontal diseases. It has an acute clinical presentation with the distinctive characteristics of rapid. Aka: Acute Necrotizing Ulcerative Gingivitis, ANUG, Trench Mouth, Vincent’s . Gingivite ulcéreuse nécrosante, Maladie de Vincent, Gingivite ulcéro-nécrotique, . MEDIDAS NO FARMACOLÓGICAS. Gingivitis ulcerativa necrosante o. Angina de Vincent (GUNA) en pacientes inmunodeprimidos,. VIH, leucemia, neutropenia.

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Periapical, mandibular and maxillary hard tissues — Bones of jaws. Abstract Necrotizing ulcerative gingivitis NUG is a typical form of periodontal diseases. Other Eagle syndrome Hemifacial hypertrophy Facial hemiatrophy Oral manifestations of systemic disease.

He had a poor plaque control without any parafunction and was a nonsmoker.

Author information Copyright and License information Disclaimer. Necrotizing ulcerative gingivitis has been observed for centuries. A bacterial infectious process affecting the gums. In developing countries, NUG may occur in children of low socioeconomic status, usually occurring with malnutrition especially inadequate protein intake and shortly after the onset of viral infections e. BMJ Case Rep Apically positioned flap Bone graft Coronally positioned flap Crown lengthening Open flap debridement Gingival graft Gingivectomy Guided bone regeneration Guided tissue regeneration Enamel matrix derivative Implant gingiviitis Lateral pedicle graft Pocket reduction surgery Socket preservation Sinus lift Subepithelial connective tissue graft Tools Curette Membrane Probe Scaler.

Treatment of the acute phase has two main objectives of therapy: Necrotizing gingivitis is part of a spectrum of disease termed necrotizing periodontal diseases.

Necrotizing Ulcerative Gingivitis

From Wikipedia, the free necrosannte. Managing patients with necrotizing ulcerative gingivitis. It is the most minor form of this spectrum, with more advanced stages being termed necrotizing periodontitis, necrotizing stomatitis and the most extreme, cancrum oris. Related Bing Images Extra: Bacteroides fragilis Tannerella forsythia Capnocytophaga canimorsus Porphyromonas gingivalis Prevotella intermedia. Archived from the original PDF on Periodontal disorders Conditions of the mucous membranes Necrosis.

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The bacterial area with a superficial fibrous mesh composed of degenerated epithelial cells, leukocytes, cellular rests, and a wide variety of bacterial cells, including rods, fusiforms, and spirochetes.

Orofacial soft tissues — Soft tissues around the mouth Actinomycosis Angioedema Basal cell carcinoma Cutaneous sinus of dental origin Cystic hygroma Gnathophyma Ludwig’s angina Macrostomia Melkersson—Rosenthal syndrome Microstomia Noma Oral Crohn’s disease Orofacial granulomatosis Perioral dermatitis Pyostomatitis vegetans. Xenophon observes sore mouth and foul smelling breath in Greek soldiers in the 4th century BC.

Acute necrotizing ulcerative gingivitis

Chlamydophila psittaci Psittacosis Chlamydophila pneumoniae. Moreover, finally, maintenance phase that allows stable outcomes.

The patient was prescribed oral antibiotic mg metronidazole every 8 h for 7 days and oral mouth rinse 0. This article is about the physiologic disorder.

enfermedades periodontales necrosantes by nadia Garcia on Prezi

A year-old male patient, with painful gingival inflammation evolving since 4 days, consulted urgently the Department of Periodontology, Faculty of Dentistry, University of Hassan II Morocco, in March Furthermore, Patient compliance is satisfactory, he has a good plaque control and respects the appointments of controls and still on maintenance phase. He had no other significant medical history or known allergies. The necrotic zone, containing disintegrated cells, together with medium- and large-size spirochetes and fusiform bacteria.

Oral mucosa — Lining of mouth.

Basal cell adenoma Canalicular adenoma Ductal papilloma Monomorphic adenoma Myoepithelioma Oncocytoma Papillary cystadenoma lymphomatosum Pleomorphic adenoma Sebaceous adenoma Malignant: Nasopalatine duct Median mandibular Median palatal Traumatic bone Osteoma Osteomyelitis Osteonecrosis Bisphosphonate-associated Neuralgia-inducing cavitational osteonecrosis Osteoradionecrosis Osteoporotic bone marrow defect Paget’s disease of bone Periapical abscess Phoenix abscess Periapical periodontitis Stafne defect Torus mandibularis.

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Clinical periodontology and implant dentistry 5th ed. Necrotizing ulcerative periodontitis NUP is where the infection leads to attachment loss, and involves only the gingiva, periodontal ligament and alveolar ligament.

It is characterized by the development of necrotic, ulcerated, and painful lesions with creation of pseudomembranes extending along the gingival margins. Orofacial soft tissues — Soft tissues around the mouth. Jean Hyacinthe Vincenta French physician working at the Paris Pasteur Institute describes a fusospirochetal infection of the pharynx and palatine tonsilscausing “ulcero-membranous pharyngitis and tonsillitis”, [5] which later became known as Vincent’s angina.

Periodontium gingivaperiodontal ligamentcementumalveolus — Gums and tooth-supporting structures Cementicle Cementoblastoma Gigantiform Cementoma Eruption cyst Epulis Pyogenic granuloma Congenital epulis Gingival enlargement Gingival cyst of the adult Gingival cyst of the newborn Gingivitis Desquamative Granulomatous Plasma cell Hereditary gingival fibromatosis Hypercementosis Hypocementosis Linear gingival erythema Necrotizing periodontal diseases Acute necrotizing ulcerative gingivitis Pericoronitis Peri-implantitis Periodontal abscess Periodontal trauma Periodontitis Aggressive As a manifestation of systemic disease Chronic Perio-endo lesion Teething.

This case report describes the diagnosis approach and the conservative management with a good outcome of NUG in a year-old male patient with no systemic disease and probable mechanism of pathogenesis of two predisposing factors involved. Actually and according to the recent data, the prevalence rate of NUG varies over a wide range from 6. The gingiva state was evaluated 2 days [ Figure 6 ] and 7 days after [ Figure 7 ] the clinical examination showed a major improvement in symptoms with almost complete resolution of the ulcerated pseudomembranous areas and reduction of erythema and swelling, and then, a subgingival debridement was conducted.